TM7SF3 controls TEAD1 splicing to prevent MASH-induced liver fibrosis-Reference-Cited by-同舟云学术

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TM7SF3 controls TEAD1 splicing to prevent MASH-induced liver fibrosis

Published:2024-05 Issue:5 Volume:36 Page:1030-1043.e7
ISSN:1550-4131
Container-title:Cell Metabolism
language:en
Short-container-title:Cell Metabolism

Author:

Isaac Roi,Bandyopadhyay Gautam,Rohm Theresa V.,Kang Sion,Wang Jinyue,Pokhrel Narayan,Sakane Sadatsugu,Zapata Rizaldy,Libster Avraham M.,Vinik Yaron,Berhan Asres,Kisseleva Tatiana,Borok Zea,Zick Yehiel,Telese Francesca,Webster Nicholas J.G.,Olefsky Jerrold M.

Funder

National Institutes of Health

Larry L Hillblom Foundation

NINDS

Janssen Pharmaceuticals Inc

Swiss National Science Foundation

Publisher

Elsevier BV

Reference54 articles.

1. From nonalcoholic steatohepatitis, metabolic dysfunction-associated fatty liver disease, to steatotic liver disease: Updates of nomenclature and impact on clinical trials;Yeh;Clin. Mol. Hepatol.,2023

2. Non-alcoholic fatty liver disease: an expanded review;Benedict;World J. Hepatol.,2017

3. Nonalcoholic Fatty Liver Disease Screening in Type 2 Diabetes Mellitus Patients in the Primary Care Setting;Vieira Barbosa;Hepatol. Commun.,2021

4. NAFLD as a continuum: from obesity to metabolic syndrome and diabetes;Godoy-Matos;Diabetol. Metab. Syndr.,2020

5. Hepatic Stellate Cell Activation and Inactivation in NASH-Fibrosis-Roles as Putative Treatment Targets?;Zisser;Biomedicines,2021

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