Fructose-1,6-bisphosphatase is a nonenzymatic safety valve that curtails AKT activation to prevent insulin hyperresponsiveness

Author:

Gu Li,Zhu Yahui,Watari Kosuke,Lee Maiya,Liu Junlai,Perez Sofia,Thai Melinda,Mayfield Joshua E.,Zhang Bichen,Cunha e Rocha Karina,Li Fuming,Kim Laura C.,Jones Alexander C.,Wierzbicki Igor H.,Liu Xiao,Newton Alexandra C.,Kisseleva Tatiana,Lee Jun Hee,Ying Wei,Gonzalez David J.,Saltiel Alan R.,Simon M. Celeste,Karin MichaelORCID

Funder

National Institutes of Health

American Cancer Society

Merck

University of Pennsylvania

University of California, San Diego

National Science Foundation

Janssen Pharmaceuticals

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Physiology

Reference45 articles.

1. Insulin signaling in health and disease;Saltiel;J. Clin. Invest.,2021

2. Regulation of hepatic glucose metabolism in health and disease;Petersen;Nat. Rev. Endocrinol.,2017

3. Insulin regulation of gluconeogenesis;Hatting;Ann. NY Acad. Sci.,2018

4. Resolving the paradox of hepatic insulin resistance;Santoleri;Cell. Mol. Gastroenterol. Hepatol.,2019

5. Interplay between FOXO, TOR, and Akt;Hay;Biochim. Biophys. Acta,2011

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