Defects in a liver-bone axis contribute to hepatic osteodystrophy disease progression-Reference-Cited by-同舟云学术

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Defects in a liver-bone axis contribute to hepatic osteodystrophy disease progression

Published:2022-03 Issue:3 Volume:34 Page:441-457.e7
ISSN:1550-4131
Container-title:Cell Metabolism
language:en
Short-container-title:Cell Metabolism

Author:

Lu Ke,Shi Tian-Shu,Shen Si-Yu,Shi Yong,Gao Hong-Liang,Wu Jing,Lu Xiang,Gao Xiang,Ju Huang-xian,Wang Wei,Cao Yi,Chen Di,Li Chao-Jun,Xue Bin,Jiang Qing

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Physiology

Reference56 articles.

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3. Both secreted and the cellular levels of BDNF attenuated due to tau hyperphosphorylation in primary cultures of cortical neurons;Atasoy;J. Chem. Neuroanat.,2017

4. Hepatic osteodystrophy: a global (re)view of the problem;Barbu;Acta Clin. Croat.,2017

5. Protein phosphatase 2A as therapeutic targets in various disease models;Baskaran;Life Sci.,2018

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2. Lecithin-cholesterol acyltransferase is a potential tumor suppressor and predictive marker for hepatocellular carcinoma metastasis;World Journal of Gastrointestinal Oncology;2024-08-15

3. Liver-based inter-organ communication: A disease perspective;Life Sciences;2024-08

4. Age-related decline in the expression of BRG1, ATM and ATR are partially reversed by dietary restriction in the livers of female mice;Biogerontology;2024-07-06

5. XAF1 promotes osteoclast apoptosis by antagonizing the XIAP-caspase axis;Journal of Orthopaedic Translation;2024-07

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