Systematic Dissection of the Metabolic-Apoptotic Interface in AML Reveals Heme Biosynthesis to Be a Regulator of Drug Sensitivity-Reference-Cited by-同舟云学术

Systematic Dissection of the Metabolic-Apoptotic Interface in AML Reveals Heme Biosynthesis to Be a Regulator of Drug Sensitivity

Published:2019-05 Issue:5 Volume:29 Page:1217-1231.e7
ISSN:1550-4131
Container-title:Cell Metabolism
language:en
Short-container-title:Cell Metabolism

Author:

Lin Kevin H.,Xie Abigail,Rutter Justine C.,Ahn Yeong-ran,Lloyd-Cowden Julia M.,Nichols Amanda G.,Soderquist Ryan S.,Koves Timothy R.,Muoio Deborah M.,MacIver Nancie J.,Lamba Jatinder K.,Pardee Timothy S.,McCall Chad M.,Rizzieri David A.,Wood Kris C.

Funder

Duke University School of Medicine

Duke Cancer Institute

NIH

Duke Medical Scientist Training Program

Duke Undergraduate Research Office

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Physiology

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3. NFS1 undergoes positive selection in lung tumours and protects cells from ferroptosis;Alvarez;Nature,2017

4. The tangled circuitry of metabolism and apoptosis;Andersen;Mol. Cell,2013

5. Heme deficiency may be a factor in the mitochondrial and neuronal decay of aging;Atamna;Proc. Natl. Acad. Sci. U S A,2002

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