SLC39A14 Is Required for the Development of Hepatocellular Iron Overload in Murine Models of Hereditary Hemochromatosis-Reference-Cited by-同舟云学术

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SLC39A14 Is Required for the Development of Hepatocellular Iron Overload in Murine Models of Hereditary Hemochromatosis

Published:2015-07 Issue:1 Volume:22 Page:138-150
ISSN:1550-4131
Container-title:Cell Metabolism
language:en
Short-container-title:Cell Metabolism

Author:

Jenkitkasemwong Supak,Wang Chia-Yu,Coffey Richard,Zhang Wei,Chan Alan,Biel Thomas,Kim Jae-Sung,Hojyo Shintaro,Fukada Toshiyuki,Knutson Mitchell D.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Physiology

Reference59 articles.

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2. Regulation of iron absorption in Hfe mutant mice;Ajioka;Blood,2002

3. Forging a field: the golden age of iron biology;Andrews;Blood,2008

4. BMP6 is a key endogenous regulator of hepcidin expression and iron metabolism;Andriopoulos;Nat. Genet.,2009

5. Non-transferrin-bound iron (NTBI) uptake by T lymphocytes: evidence for the selective acquisition of oligomeric ferric citrate species;Arezes;PLoS ONE,2013

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