Specific Roles of the p110α Isoform of Phosphatidylinsositol 3-Kinase in Hepatic Insulin Signaling and Metabolic Regulation-Reference-Cited by-同舟云学术

Specific Roles of the p110α Isoform of Phosphatidylinsositol 3-Kinase in Hepatic Insulin Signaling and Metabolic Regulation

Published:2010-03 Issue:3 Volume:11 Page:220-230
ISSN:1550-4131
Container-title:Cell Metabolism
language:en
Short-container-title:Cell Metabolism

Author:

Sopasakis Victoria Rotter,Liu Pixu,Suzuki Ryo,Kondo Tatsuya,Winnay Jonathon,Tran Thien T.,Asano Tomoichiro,Smyth Graham,Sajan Mini P.,Farese Robert V.,Kahn C. Ronald,Zhao Jean J.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Physiology

Reference45 articles.

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3. Proliferative defect and embryonic lethality in mice homozygous for a deletion in the p110alpha subunit of phosphoinositide 3-kinase;Bi;J. Biol. Chem.,1999

4. Early embryonic lethality in mice deficient in the p110beta catalytic subunit of PI 3-kinase;Bi;Mamm. Genome,2002

5. Phosphoinositide 3-kinase catalytic subunit deletion and regulatory subunit deletion have opposite effects on insulin sensitivity in mice;Brachmann;Mol. Cell. Biol.,2005

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