ATF4 mediation of NF1 functions in osteoblast reveals a nutritional basis for congenital skeletal dysplasiae-Reference-Cited by-同舟云学术

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ATF4 mediation of NF1 functions in osteoblast reveals a nutritional basis for congenital skeletal dysplasiae

Published:2006-12 Issue:6 Volume:4 Page:441-451
ISSN:1550-4131
Container-title:Cell Metabolism
language:en
Short-container-title:Cell Metabolism

Author:

Elefteriou Florent,Benson M. Douglas,Sowa Hideaki,Starbuck Michael,Liu Xiuyun,Ron David,Parada Luis F.,Karsenty Gerard

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Physiology

Reference52 articles.

1. Is osseous dysplasia a primary feature of neurofibromatosis 1 (NF1)?;Alwan;Clin. Genet.,2005

2. Changes in collagen biosynthesis, types, and mechanics of aorta in hypertensive rats;Bashey;J. Lab. Clin. Med.,1989

3. Haploinsufficiency of Sox9 results in defective cartilage primordia and premature skeletal mineralization;Bi;Proc. Natl. Acad. Sci. USA,2001

4. A twist code determines the onset of osteoblast differentiation;Bialek;Dev. Cell,2004

5. Mouse alpha1(I)-collagen promoter is the best known promoter to drive efficient Cre recombinase expression in osteoblast;Dacquin;Dev. Dyn.,2002

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5. Generation of heterozygous and homozygous NF1 lines from human-induced pluripotent stem cells using CRISPR/Cas9 to investigate bone defects associated with neurofibromatosis type 1;Frontiers in Cell and Developmental Biology;2024-02-28

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