A mouse model for high-efficient Flp-recombinase-mediated genetic manipulation in the pancreas

Author:

Zhu XiaohuiORCID,Chen JiaxiangORCID,Wang Bin,Wang Lingxiang,Wang Jiale,Feng DongfengORCID,Yang YanORCID,Wang Oliver,Haddock Ashley N.,Wang Ying,Ji Baoan,Bi YanORCID

Funder

U.S. Department of Defense

Mayo Clinic

Publisher

Elsevier BV

Subject

Gastroenterology,Hepatology,Endocrinology, Diabetes and Metabolism

Reference9 articles.

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2. Genetically engineered mouse models of pancreatic cancer: the KPC model (LSL-Kras(G12D/+) ;LSL-Trp53(R172H/+) ;Pdx-1-Cre), its variants, and their application in immuno-oncology drug discovery;Lee;Curr Protoc Pharmacol,2016

3. Pancreas-specific Cre driver lines and considerations for their prudent use;Magnuson;Cell Metabol,2013

4. Trp53R172H and KrasG12D cooperate to promote chromosomal instability and widely metastatic pancreatic ductal adenocarcinoma in mice;Hingorani;Cancer Cell,2005

5. Genetic progression of pancreatic cancer;Cowan;Cancer J,2014

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