Mouse model of PRSS1 p.R122H-related hereditary pancreatitis highlights context-dependent effect of autolysis-site mutation-Reference-Cited by-同舟云学术

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Mouse model of PRSS1 p.R122H-related hereditary pancreatitis highlights context-dependent effect of autolysis-site mutation

Published:2023-03 Issue:2 Volume:23 Page:131-142
ISSN:1424-3903
Container-title:Pancreatology
language:en
Short-container-title:Pancreatology

Author:

Jancsó Zsanett,Morales Granda Nataly C.^ORCID,Demcsák Alexandra^ORCID,Sahin-Tóth Miklós^ORCID

Funder

NIDDK

Missouri University of Science and Technology

National Institutes of Health

U.S. Department of Defense

Publisher

Elsevier BV

Subject

Gastroenterology,Hepatology,Endocrinology, Diabetes and Metabolism

Reference42 articles.

1. Hereditary pancreatitis is caused by a mutation in the cationic trypsinogen gene;Whitcomb;Nat Genet,1996

2. Human cationic trypsinogen (PRSS1) variants and chronic pancreatitis;Németh;Am J Physiol Gastrointest Liver Physiol,2014

3. Genetic risk in chronic pancreatitis: the trypsin-dependent pathway;Hegyi;Dig Dis Sci,2017

4. Genetics, cell biology, and pathophysiology of pancreatitis;Mayerle;Gastroenterology,2019

5. On some autolyzed derivatives of bovine trypsin;Maroux;Biochim Biophys Acta,1969

Cited by 3 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Heterozygous Spink1 c.194+2T>C mutation promotes chronic pancreatitis after acute attack in mice;Pancreatology;2024-08

2. Understanding the toxicity mechanism of gelsemine in zebrafish;Comparative Biochemistry and Physiology Part C: Toxicology & Pharmacology;2024-06

3. Classification of PRSS1 variants responsible for chronic pancreatitis: An expert perspective from the Franco-Chinese GREPAN Study Group;Pancreatology;2023-08

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