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Slow changes of tyrosine hydroxylase gene expression in dopaminergic brain neurons after neurotoxin lesioning: a model for neuron aging

  • Published:1992-03 Issue:1-2 Volume:13 Page:63-73
  • ISSN:0169-328X
  • Container-title:Molecular Brain Research
  • language:en
  • Short-container-title:Molecular Brain Research

Author:

Pasinetti G.M.,Osterburg H.H.,Kelly A.B.,Kohama S.,Morgan D.G.,Reinhard J.F.,Stellwagen R.H.,Finch C.E.

Publisher

Elsevier BV

Subject

Cellular and Molecular Neuroscience,Molecular Biology

Reference78 articles.

1. Hyperactivity of remaining dopaminergic neurones after partial destruction of the nigro-striatal dopaminergic system in the rat;Agid;Nature,1973

2. Biochemistry of neurotransmitters in Parkinson's disease;Agid,1987

3. The crossed mesostriatal projection: neurochemistry and developmental response to lesion;Altar;Brain Res.,1983

4. Transneuronal regulation of tyrosine hydroxylase expression in olfactory bulb of mouse and rat;Baker;J. Neurosci.,1983

5. Effects of cold exposure on rat adrenal tyrosine hydroxylase: an analysis of RNA, protein, enzyme activity, and cofactor levels;Baruchin;J. Neurochem.,1990
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