Analysis of c-Jun N-Terminal Kinase Regulation and Function
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Publisher
Elsevier
Reference35 articles.
1. Signal transduction by the c-Jun N-terminal kinase (JNK) — from inflammation to development
2. The stress-activated protein kinase pathways
3. Signal Transduction through MAP Kinase Cascades
4. JNK1: A protein kinase stimulated by UV light and Ha-Ras that binds and phosphorylates the c-Jun activation domain
5. Pro-inflammatory Cytokines and Environmental Stress Cause p38 Mitogen-activated Protein Kinase Activation by Dual Phosphorylation on Tyrosine and Threonine
Cited by 5 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Enhanced JNK activation by NESK without kinase activity upon caspase-mediated cleavage during apoptosis;Cellular Signalling;2005-11
2. A multiplexed homogeneous fluorescence-based assay for protein kinase activity in cell lysates;Nature Methods;2005-03-23
3. Glucose Utilization Is Essential for Hypoxia-Inducible Factor 1α-Dependent Phosphorylation of c-Jun;Molecular and Cellular Biology;2004-05-15
4. Long-term activation of SAPK/JNK, p38 kinase and fas-L expression by cisplatin is attenuated in human carcinoma cells that acquired drug resistance;International Journal of Cancer;2004
5. Inhibition of the p38 pathway upregulates macrophage JNK and ERK activities, and the ERK, JNK, and p38 MAP kinase pathways are reprogrammed during differentiation of the murine myeloid M1 cell line;Journal of Cellular Biochemistry;2002
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