Inhibitors of membrane lipid metabolism enhance complement-mediated nucleated cell killing through distinct mechanisms

Author:

Papadimitriou J.C.,Carney D.F.,Shin M.L.

Publisher

Elsevier BV

Subject

Molecular Biology,Immunology

Reference39 articles.

1. Elimination of terminal complement complexes in the plasma membrane of nucleated cells: influence of extracellular Ca2+ and association with cellular Ca2+;Carney;J. Immun.,1986

2. Elimination of terminal complement intermediates from the plasma membrane of nucleated cells: the rate of disappearance differs for cells carrying C5b-7 or C5b-8 or a mixture of C5b-8 with a limited number of C5b-9;Carney;J. Immun.,1985

3. Terminal complement complexes generate multiple signal messengers: protein kinase C but not cAMP is involved in rapid elimination of TCC from the surface of Ehrlich cells;Carney;J. Immun.,1990

4. Role of lipid fatty acid composition and membrane fluidity in the resistance of Acholeplasma laidlawii to complement-mediated killing;Dahl;J. Immun.,1979

5. Induction of prostanoid synthesis in human platelets by the late complement components C5b-9 and channel forming antibiotic nystatin: inhibition of the reayclation of liberated arachidonic acid;Hänsch;J. Immun.,1985

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