Stress activated protein kinases, a novel family of mitogen-activated protein kinases, are heterogeneously expressed in the adult rat brain and differentially distributed from extracellular-signal-regulated protein kinases-Reference-Cited by-同舟云学术

Stress activated protein kinases, a novel family of mitogen-activated protein kinases, are heterogeneously expressed in the adult rat brain and differentially distributed from extracellular-signal-regulated protein kinases

Published:1995-12 Issue:4 Volume:69 Page:1103-1110
ISSN:0306-4522
Container-title:Neuroscience
language:en
Short-container-title:Neuroscience

Author:

Carletti R.,Tacconi S.,Bettini E.,Ferraguti F.

Publisher

Elsevier BV

Subject

General Neuroscience

Reference24 articles.

1. Stimulation of protein tyrosine phosphorylation by NMDA receptor activation;Bading;Science,1991

2. Diversity in function and regulation of MAP kinase pathway;Blumer;Trends biochem. Sci.,1994

3. An insulin-stimulated protein kinase similar to yeast kinases involved in cell cycle control;Boulton;Science,1990

4. ERKs: a family of protein-serine/threonine kinases that are activated and tyrosine phosphorylated in response to insulin and NGF;Boulton;Cell,1991

5. Parallel signal processing among mammalian MAPKs;Cano;Trends biochem. Sci.,1995

Cited by 45 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Inhibition of mTOR protects against skeletal muscle and kidney injury following hindlimb ischemia-reperfusion in rats by regulating MERK1/ERK1/2 activity.;Cukurova Medical Journal;2022-03-31

2. Mechanistic insight on the role of leukotriene receptors in ischemic–reperfusion injury;Pharmacological Reports;2021-04-05

3. Detection of MAPK signal transduction proteins in an ischemia/reperfusion model of mouse intestine using in vivo cryotechnique;Histochemistry and Cell Biology;2013-06-23

4. Intracellular Signaling MAPK Pathway After Cerebral Ischemia–Reperfusion Injury;Neurochemical Research;2012-03-20

5. MAPK, β-amyloid and synaptic dysfunction: the role of RAGE;Expert Review of Neurotherapeutics;2009-11