Selective vulnerability of hippocampal CA1 neurons cannot be explained in terms of an increase in glutamate concentration during ischemia in the gerbil: Brain microdialysis study-Reference-Cited by-同舟云学术

Selective vulnerability of hippocampal CA1 neurons cannot be explained in terms of an increase in glutamate concentration during ischemia in the gerbil: Brain microdialysis study

Published:1992-05 Issue:2 Volume:48 Page:307-313
ISSN:0306-4522
Container-title:Neuroscience
language:en
Short-container-title:Neuroscience

Author:

Mitani A.,Andou Y.,Kataoka K.

Publisher

Elsevier BV

Subject

General Neuroscience

Reference36 articles.

1. Elevation of the extracellular concentrations of glutamate and aspartate in rat hippocampus during transient cerebral ischemia monitored by intracerebral microdialysis;Benveniste;J. Neurochem.,1984

2. Calcium accumulation by glutamate receptor activation is involved in hippocampal cell damage after ischemia;Benveniste;Acta neurol. scand.,1988

3. Ischemic damage in hippocampal CA1 is dependent on glutamate release and intact innervation from CA3;Benveniste;J. cerebr. Blood Flow Metab.,1989

4. Cerebral hypoxia;Brierley,1976

5. Ischemia-induced neuronal cell death, calcium accumulation, and glial response in the hippocampus of the Mongolian gerbil and protection by propentofylline (HWA 285);DeLeo;J. cerebr. Blood Flow Metab.,1987

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