The yeast mitochondrial permeability transition is regulated by reactive oxygen species, endogenous Ca2+ and Cpr3, mediating cell death-Reference-Cited by-同舟云学术

The yeast mitochondrial permeability transition is regulated by reactive oxygen species, endogenous Ca2+ and Cpr3, mediating cell death

Published:2018-12 Issue:12 Volume:1859 Page:1313-1326
ISSN:0005-2728
Container-title:Biochimica et Biophysica Acta (BBA) - Bioenergetics
language:en
Short-container-title:Biochimica et Biophysica Acta (BBA) - Bioenergetics

Author:

Kamei Yoshiko,Koushi Masami,Aoyama Yasunori,Asakai Rei

Publisher

Elsevier BV

Subject

Cell Biology,Biochemistry,Biophysics

Reference77 articles.

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2. Inhibition by cyclosporin A of a Ca2+-dependent pore in heart mitochondria activated by inorganic phosphate and oxidative stress;Crompton;Biochem. J.,1988

3. Cyclosporin A is a potent inhibitor of the inner membrane permeability transition in liver mitochondria;Broekemeier;J. Biol. Chem.,1989

4. Inhibition of Ca2+-induced large-amplitude swelling of liver and heart mitochondria by cyclosporin is probably caused by the inhibitor binding to mitochondrial-matrix peptidyl-prolyl cis-trans isomerase and preventing it interacting with the adenine nucleotide translocase

5. Recruitment of mitochondrial cyclophilin to the mitochondrial inner membrane under conditions of oxidative stress that enhance the opening of a calcium-sensitive non-specific channel;Connern;Biochem. J.,1994

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