ROS production in brown adipose tissue mitochondria: The question of UCP1-dependence-Reference-Cited by-同舟云学术

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ROS production in brown adipose tissue mitochondria: The question of UCP1-dependence

Published:2014-12 Issue:12 Volume:1837 Page:2017-2030
ISSN:0005-2728
Container-title:Biochimica et Biophysica Acta (BBA) - Bioenergetics
language:en
Short-container-title:Biochimica et Biophysica Acta (BBA) - Bioenergetics

Author:

Shabalina Irina G.,Vrbacký Marek,Pecinová Alena,Kalinovich Anastasia V.,Drahota Zdeněk,Houštěk Josef,Mráček Tomáš,Cannon Barbara,Nedergaard Jan

Funder

Swedish Research Council Formas

European Union

Knut and Alice Wallenberg Foundation

Stockholm University Academic

Ministry of Education, Youth and Sports of the Czech Republic

Grant Agency of the Czech Republic

Publisher

Elsevier BV

Subject

Cell Biology,Biochemistry,Biophysics

Reference72 articles.

1. The role of UCP 1 in production of reactive oxygen species by mitochondria isolated from brown adipose tissue;Dlaskova;Biochim. Biophys. Acta,2010

2. Uncoupling protein 1 decreases superoxide production in brown adipose tissue mitochondria;Oelkrug;J. Biol. Chem.,2010

3. UCP1 and defense against oxidative stress. 4-Hydroxy-2-nonenal effects on brown fat mitochondria are uncoupling protein 1-independent;Shabalina;J. Biol. Chem.,2006

4. Crucial yet divergent roles of mitochondrial redox state in skeletal muscle vs. brown adipose tissue energetics;Mailloux;FASEB J.,2012

5. Brown adipose tissue mitochondria oxidizing fatty acids generate high levels of reactive oxygen species irrespective of the uncoupling protein-1 activity state;Schonfeld;Biochim. Biophys. Acta,2012

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