Modulation of macrophage apoptosis by antimycobacterial therapy: physiological role of apoptosis in the control of Mycobacterium tuberculosis

Author:

Gil Diana,Garcia Luis F.,Rojas Mauricio

Publisher

Elsevier BV

Subject

Pharmacology,Toxicology

Reference56 articles.

1. Hepatotoxic effects of tuberculosis therapy;Ali;Postgrad. Med.,1996

2. American Medical Association, 1994. Antimycobacterial drugs, in: American Medical Drugs Evaluation Annual 1995, Association (Ed.), Albany, NY, pp. 1685–1726

3. Inhibition of virulent Mycobacterium tuberculosis by Bcgr and Bcgs macrophages correlates with nitric oxide production;Arias;J. Infect. Dis.,1997

4. Balcewicz-Sablinska, M. Katarzyna, Keane, J., Kornfeld, H., Remold, H.G., 1998. Pathogenic Mycobacterium tuberculosis evades apoptosis of host macrophages by release of host TNF-R2, resulting in inactivation of TNF-alpha. J. Immunol. 161, 2636–2641

5. Calcium metabolism during rifampicin and isoniazid therapy for tuberculosis;Brodie;J. R. Soc. Med.,1982

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