Overexpression of hepatic serum amyloid A1 in mice increases IL-17-producing innate immune cells and decreases bone density-Reference-Cited by-同舟云学术

Overexpression of hepatic serum amyloid A1 in mice increases IL-17-producing innate immune cells and decreases bone density

Published:2021-01 Issue: Volume:296 Page:100595
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Choi Minjee,Park Song,Yi Jun Koo^ORCID,Kwon Wookbong^ORCID,Jang Soyoung^ORCID,Kim Si-Yong^ORCID,Yu Wookyung,Kim Myoung Ok,Ryoo Zae Young,Choi Seong-Kyoon

Funder

Ministry of Science, ICT and Future Planning

National Research Foundation of Korea

DGIST

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference31 articles.

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4. An IL-23R/IL-22 circuit regulates epithelial serum amyloid A to promote local effector Th17 responses;Sano;Cell,2015

5. Serum amyloid A promotes lung neutrophilia by increasing IL-17A levels in the mucosa and gammadelta T cells;Anthony;Am. J. Respir. Crit. Care Med.,2013

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