PP2A inhibitor SET promotes mTORC1 and Bmi1 signaling through Akt activation and maintains the colony-formation ability of cancer cells-Reference-Cited by-同舟云学术

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PP2A inhibitor SET promotes mTORC1 and Bmi1 signaling through Akt activation and maintains the colony-formation ability of cancer cells

Published:2024-01 Issue:1 Volume:300 Page:105584
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Kohyanagi Naoki,Kitamura Nao,Ikeda Shunta,Shibutani Shusaku,Sato Koichi,Ohama Takashi

Funder

Japan Society for the Promotion of Science

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference48 articles.

1. Therapeutic targeting of PP2A;O’Connor;Int. J. Biochem. Cell Biol.,2018

2. Overexpression of SET is a recurrent event associated with poor outcome and contributes to protein phosphatase 2A inhibition in acute myeloid leukemia;Cristóbal;Haematologica,2012

3. Involvement of SET in the Wnt signaling pathway and the development of human colorectal cancer;Dong;Oncol. Lett.,2014

4. Stemness is enhanced in gastric cancer by a SET/PP2A/E2F1 Axis;Enjoji;Mol. Cancer Res.,2018

5. Targeting inhibitors of the tumor suppressor PP2A for the treatment of pancreatic cancer;Farrell;Mol. Cancer Res.,2014

Cited by 2 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Paradoxical action of PP2A inhibition and its potential for therapeutic sensitization;Journal of Cellular Physiology;2024-08-16

2. Hyperphosphorylated Tau Targeting Human Serum Albumin Fusion Protein as Therapeutics for Alzheimer's Diseases;2024

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