Oxidative stress–induced autonomous activation of the calcium/calmodulin-dependent kinase II involves disulfide formation in the regulatory domain-Reference-Cited by-同舟云学术

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Oxidative stress–induced autonomous activation of the calcium/calmodulin-dependent kinase II involves disulfide formation in the regulatory domain

Published:2022-11 Issue:11 Volume:298 Page:102579
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Rocco-Machado Nathália,Lai Lo,Kim Geumsoo,He Yi,Luczak Elizabeth D.^ORCID,Anderson Mark E.,Levine Rodney L.

Funder

National Institutes of Health

Johns Hopkins School of Medicine

National Heart Lung and Blood Institute Division of Intramural Research

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference56 articles.

1. Neuronal CA2+/calmodulin-dependent protein kinase II: the role of structure and autoregulation in cellular function;Hudmon;Annu. Rev. Biochem.,2002

2. CaMKII in vascular signalling: “friend or foe”?;Ebenebe;Heart Lung Circ.,2018

3. CaMKII in regulation of cell death during myocardial reperfusion injury;Yang;Front. Mol. Biosci.,2021

4. CaMKIIdelta isoforms differentially affect calcium handling but similarly regulate HDAC/MEF2 transcriptional responses;Zhang;J. Biol. Chem.,2007

5. Role of Ca2+/calmodulin-dependent protein kinase (CaMK) in excitation-contraction coupling in the heart;Maier;Cardiovasc. Res.,2007

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4. Nitric Oxide Modulates Ca ²⁺ Leak and Arrhythmias via S-Nitrosylation of CaMKII;Circulation Research;2023-12-08

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