Dipeptidyl peptidase 4 contributes to Alzheimer’s disease–like defects in a mouse model and is increased in sporadic Alzheimer’s disease brains-Reference-Cited by-同舟云学术

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Dipeptidyl peptidase 4 contributes to Alzheimer’s disease–like defects in a mouse model and is increased in sporadic Alzheimer’s disease brains

Published:2021-08 Issue:2 Volume:297 Page:100963
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Valverde Audrey,Dunys Julie,Lorivel Thomas,Debayle Delphine,Gay Anne-Sophie,Caillava Céline,Chami Mounia,Checler Frédéric

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference59 articles.

1. Alzheimer's disease: The amyloid cascade hypothesis;Hardy;Science,1992

2. Processing of the b-amyloid precursor protein and its regulation in Alzheimer's disease;Checler;J. Neurochem.,1995

3. A critical appraisal of amyloid-beta-targeting therapies for Alzheimer disease;Panza;Nat. Rev. Neurol.,2019

4. Major clinical trials failed the amyloid hypothesis of Alzheimer's disease;Huang;J. Am. Geriatr. Soc.,2019

5. The case for rejecting the amyloid cascade hypothesis;Herrup;Nat. Neurosci.,2015

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