CTLA-4 polymorphism in the pathogenesis of chronic spontaneous autoreactive urticaria-Reference-Cited by-同舟云学术

CTLA-4 polymorphism in the pathogenesis of chronic spontaneous autoreactive urticaria

Published:2014-05 Issue:3 Volume:42 Page:241-244
ISSN:0301-0546
Container-title:Allergologia et Immunopathologia
language:en
Short-container-title:Allergologia et Immunopathologia

Author:

Brzoza Z.,Grzeszczak W.,Rogala B.,Trautsolt W.,Moczulski D.

Publisher

Codon Publications

Subject

General Medicine,Immunology and Allergy,Immunology,Pulmonary and Respiratory Medicine

Reference27 articles.

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4. The genetic susceptibility to Graves’ disease;Tomer;Baillieres Clin Endocrinol Metab,1997

5. Co-stimulation in T cell responses;Chambers;Curr Opin Immunol,1997

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1. Lack evidence of association of PTPN‐22 and CTLA‐4 polymorphisms with risk of chronic spontaneous urticaria in Han Chinese population;Journal of Cosmetic Dermatology;2021-10-07

2. Interleukin 1 Gene Polymorphisms Presumably Participate in the Pathogenesis of Chronic Spontaneous Autoreactive Urticaria;Journal of Interferon & Cytokine Research;2020-10-01

3. Immunoglobulin E-Mediated Autoimmunity;Frontiers in Immunology;2018-04-09

4. Inducible T-cell costimulator (ICOS ) and CD28 polymorphisms possibly play a role in the pathogenesis of chronic autoreactive urticaria;Clinical and Experimental Dermatology;2017-09-20

5. Associations of single nucleotide polymorphisms of PTPN22 and Ctla4 genes with the risk of allergic rhinitis in a Chinese Han population;Human Immunology;2017-02