β-Amyloid peptide-induced morphological changes coincide with increased K+ and Cl− channel activity in rat cortical astrocytes

Author:

Jalonen Tuula O,Charniga Carol J,Wielt Dustin B

Publisher

Elsevier BV

Subject

Developmental Biology,Clinical Neurology,Molecular Biology,General Neuroscience

Reference56 articles.

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2. Arispe, N., Pollard, H.B. and Rojas, E., β-Amyloid Ca2+-channel hypothesis for neuronal death in Alzheimer disease, Mol. Cell. Biochem., 140 (1994) 119–125.

3. Arispe, N., Pollard, H.B. and Rojas, E., Zn2+ interaction with Alzheimer amyloid β protein calcium channels, Proc. Natl. Acad. Sci. USA, 93 (1996) 1710–1715.

4. Behl, C., Davis, J.B., Klier, F.G. and Schubert, D., Amyloid β peptide induces necrosis rather than apoptosis, Brain Res., 645 (1994) 253–264.

5. Brorson, J.R., Bindokas, V.P., Iwama, T., Marcuccilli, C.J., Chisholm, J.C. and Miller, R.J., The Ca2+ influx induced by β-amyloid peptide(25-35) in cultured hippocampal neurons results from network excitation, J. Neurobiol., 26 (1995) 325–338.

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