Calretinin and calbindin D-28k delay the onset of cell death after excitotoxic stimulation in transfected P19 cells-Reference-Cited by-同舟云学术

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Calretinin and calbindin D-28k delay the onset of cell death after excitotoxic stimulation in transfected P19 cells

Published:2001-08 Issue:1-2 Volume:909 Page:145-158
ISSN:0006-8993
Container-title:Brain Research
language:en
Short-container-title:Brain Research

Author:

D’Orlando Cristina,Fellay Benoı̂t,Schwaller Beat,Salicio Valérie,Bloc Alain,Gotzos Vassilis,Celio Marco R

Publisher

Elsevier BV

Subject

Developmental Biology,Neurology (clinical),Molecular Biology,General Neuroscience

Reference91 articles.

1. The effects of artificial calcium buffers on calcium responses and glutamate-mediated excitotoxicity in cultured hippocampal neurons;Abdel-Hamid;Neuroscience,1997

2. Vulnerability of midbrain dopaminergic neurons in calbindin-D28k-deficient mice: lack of evidence for a neuroprotective role of endogenous calbindin in MPTP-treated and weaver mice;Airaksinen;Eur. J. Neurosci.,1997

3. Calcium-binding proteins: selective markers of nerve cells;Andressen;Cell Tissue Res.,1993

4. Changes in shape and motility of cells transfected with parvalbumin cDNA;Andressen;Exp. Cell Res.,1995

5. Glutamate-induced neuronal death: a succession of necrosis or apoptosis depending on mitochondrial function;Ankarcrona;Neuron,1995

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