Steroid-inhibition of [3H]γ-hydroxybutyric acid (GHB) binding in thalamic relay nuclei increases during absence seizures-Reference-Cited by-同舟云学术

Steroid-inhibition of [3H]γ-hydroxybutyric acid (GHB) binding in thalamic relay nuclei increases during absence seizures

Published:1998-12 Issue:2 Volume:813 Page:343-350
ISSN:0006-8993
Container-title:Brain Research
language:en
Short-container-title:Brain Research

Author:

Banerjee Pradeep K,Liu C.C,Snead O.Carter

Publisher

Elsevier BV

Subject

Developmental Biology,Clinical Neurology,Molecular Biology,General Neuroscience

Reference24 articles.

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3. γ-Hyroxybutyric acid induced spike and wave discharges in rats: relation to high affinity [3H]γ-hydroxybutyric acid binding sites in the thalamus and cortex;Banerjee;Neuroscience,1993

4. Neurosteroid modulation of [3H]γ-hydroxybutyric acid (GHB) and [3H]GABA(B) binding in the cortex and thalamus: further evidence that GHB and GABA(B) binding sites are not similar;Banerjee;Epilepsia,1995

5. P.K. Banerjee, O.C. Snead, Neuroactive steroids exacerbate γ-hydroxybutyric acid-induced absence-like seizures in rats, Eur. J. Pharmacol., 1998, in press.

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1. Pharmacologic Models of Generalized Absence Seizures in Rodents;Models of Seizures and Epilepsy;2006

2. Metabolic GHB precursor succinate binds to γ-hydroxybutyrate receptors: Characterization of human basal ganglia areasnucleus accumbens andglobus pallidus;Journal of Neuroscience Research;2006

3. ??-Hydroxybutyric Acid;Toxicological Reviews;2004

4. 3 alpha-hydroxysteroid dehydrogenase in animal and human tissues;Biochemistry (Moscow);2001