Decreased seizure threshold and more rapid rate of kindling in rats with cortical malformation induced by prenatal treatment with methylazoxymethanol.
Author:
Publisher
Elsevier BV
Subject
Developmental Biology,Neurology (clinical),Molecular Biology,General Neuroscience
Reference13 articles.
1. Microencephalic rats as a model for cognitive disorders;Balduini;Clin. Neuropharmacol.,1986
2. Flurothyl seizure susceptibility in rats following prenatal methylazoxymethanol treatment;Baraban;Epilepsy Res.,1996
3. Neuronal migration disorders: Heterotopic neocortical neurons in CA1 form a bridge between the hippocampus and the neocortex;Chevassus-au-Louis;Proc. Natl. Acad. Sci. U.S.A.,1998
4. Neocortex in the hippocampus: morpho-functional analysis of CA1 heterotopias after prenatal treatment with methylazoxymethanol in rats;Chevassus-au-Louis;J. Comp. Neurol.,1998
5. Telencephalic cytoarchitectonics in the brains of rats with graded degrees of micrencephaly;Dambska;Acta Neuropathol.,1982
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