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Stimulus duration modulates the interaction between opioids and nitric oxide in hypoxic pial artery dilation

  • Published:1999-04 Issue:1-2 Volume:825 Page:68-74
  • ISSN:0006-8993
  • Container-title:Brain Research
  • language:en
  • Short-container-title:Brain Research

Author:

Armstead William M.

Publisher

Elsevier BV

Subject

Developmental Biology,Clinical Neurology,Molecular Biology,General Neuroscience

Reference15 articles.

1. Opioids and nitric oxide contribute to hypoxia-induced pial artery vasodilation in the newborn pig;Armstead;Am. J. Physiol.,1995

2. The contribution of δ1 and δ2-opioid receptors to hypoxia-induced pial artery dilation in the newborn pig;Armstead;J. Cereb. Blood Flow Metab.,1995

3. Role of opioids in hypoxic pial artery dilation is stimulus duration dependent;Armstead;Am. J. Physiol.,1998

4. Relationship between NO, the KATP channel and opioids in hypoxic pial artery dilation;Armstead;Am. J. Physiol.,1998

5. Effects of indomethacin on brain blood flow and cerebral metabolism in hypoxic newborn piglets;Coyle;Am. J. Physiol.,1993

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1. On the mechanisms of brain blood flow regulation during hypoxia;The Journal of Physiology;2024-06-06

2. A Neurovascular Perspective for Long-Term Changes After Brain Trauma;Translational Stroke Research;2011-11-10

3. How mental stress affects endothelial function;Pflügers Archiv - European Journal of Physiology;2011-09-23

4. Opioid system functional regulation in neurological disease management;Journal of Neuroscience Research;2010-08-23

5. Hypoxic regulation of the fetal cerebral circulation;Journal of Applied Physiology;2006-02
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