PU.1 is dispensable to block erythroid differentiation in Friend erythroleukemia cells
Author:
Publisher
Elsevier BV
Subject
Cancer Research,Oncology,Hematology
Reference51 articles.
1. Friend virus-induced erythroleukemia and the multistage nature of cancer;Ben-David;Cell,1991
2. Cell-free transmission in adult Swiss mice of a disease having the character of a leukemia;Friend;J Exp Med,1957
3. Deregulation of erythropoiesis by the Friend spleen focus-forming virus;Ruscetti;Int J Biochem Cell Biol,1999
4. Activation of cell growth by binding of Friend spleen focus-forming virus gp55 glycoprotein to the erythropoietin receptor;Li;Nature,1990
5. The envelope glycoprotein of friend spleen focus-forming virus covalently interacts with and constitutively activates a truncated form of the receptor tyrosine kinase Stk;Nishigaki;J Virol,2001
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1. CRISPR/Cas9-mediated deletion of the Wiskott-Aldrich syndrome locus causes actin cytoskeleton disorganization in murine erythroleukemia cells;PeerJ;2019-01-16
2. Mbd2-CP2c loop drives adult-type globin gene expression and definitive erythropoiesis;Nucleic Acids Research;2018-03-14
3. Differential gene expression analysis by RNA-seq reveals the importance of actin cytoskeletal proteins in erythroleukemia cells;PeerJ;2017-06-27
4. DNA methylation-mediated silencing of PU.1 in leukemia cells resistant to cell differentiation;SpringerPlus;2013-08-21
5. Emodin can induce K562 cells to erythroid differentiation and improve the expression of globin genes;Molecular and Cellular Biochemistry;2013-06-07
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