A novel K509I mutation of KIT identified in familial mastocytosis—in vitro and in vivo responsiveness to imatinib therapy-Reference-Cited by-同舟云学术

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A novel K509I mutation of KIT identified in familial mastocytosis—in vitro and in vivo responsiveness to imatinib therapy

Published:2006-04 Issue:4 Volume:30 Page:373-378
ISSN:0145-2126
Container-title:Leukemia Research
language:en
Short-container-title:Leukemia Research

Author:

Zhang Ling Yan,Smith Matthew L.,Schultheis Beate,Fitzgibbon Jude,Lister T. Andrew,Melo Junia V.,Cross Nicholas C.P.,Cavenagh Jamie D.

Publisher

Elsevier BV

Subject

Cancer Research,Oncology,Hematology

Reference25 articles.

1. Tyrosine kinase oncogenes in normal hematopoiesis and hematological disease;Scheijen;Oncogene,2002

2. Constitutive activation of c-kit in FMA3 murine mastocytoma cells caused by deletion of seven amino acids at the juxtamembrane domain;Tsujimura;Blood,1996

3. Identification of mutations in the coding sequence of the proto-oncogene c-kit in a human mast cell leukemia cell line causing ligand-independent activation of c-kit product;Furitsu;J Clin Invest,1993

4. Identification of activating c-kit mutations in adult-, but not in childhood-onset indolent mastocytosis: a possible explanation for divergent clinical behavior;Buttner;J Invest Dermatol,1998

5. Signal transducer and activator of transcription 3 activation is required for Asp(816) mutant c-Kit-mediated cytokine-independent survival and proliferation in human leukemia cells;Ning;Blood,2001

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