Combination of IAP Antagonists and TNF-α-Armed Oncolytic Viruses Induce Tumor Vascular Shutdown and Tumor Regression-Reference-Cited by-同舟云学术

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Combination of IAP Antagonists and TNF-α-Armed Oncolytic Viruses Induce Tumor Vascular Shutdown and Tumor Regression

Published:2018-09 Issue: Volume:10 Page:28-39
ISSN:2372-7705
Container-title:Molecular Therapy - Oncolytics
language:en
Short-container-title:Molecular Therapy - Oncolytics

Author:

Beug Shawn T.,Pichette Stephanie J.,St-Jean Martine,Holbrook Janelle,Walker Danielle E.,LaCasse Eric C.,Korneluk Robert G.

Funder

Canadian Cancer Society Research Institute (CCSRI)

Brain Canada

Canadian Institutes of Health Research

Ottawa Regional Cancer Foundation (ORCF)

Ottawa Kiwanis Medical Foundation

Publisher

Elsevier BV

Subject

Pharmacology (medical),Cancer Research,Oncology,Molecular Medicine

Reference47 articles.

1. Targeting IAP proteins for therapeutic intervention in cancer;Fulda;Nat. Rev. Drug Discov.,2012

2. Promises and Challenges of Smac Mimetics as Cancer Therapeutics;Fulda;Clin. Cancer Res.,2015

3. Both cIAP1 and cIAP2 regulate TNFalpha-mediated NF-kappaB activation;Mahoney;Proc. Natl. Acad. Sci. USA,2008

4. The inhibitors of apoptosis (IAPs) as cancer targets;Hunter;Apoptosis,2007

5. Activation of IKK by TNFalpha requires site-specific ubiquitination of RIP1 and polyubiquitin binding by NEMO;Ea;Mol. Cell,2006

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