Genetics Leads the Way to the Accomplices of Presenilins
Author:
Publisher
Elsevier BV
Subject
Developmental Biology,Cell Biology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology
Reference10 articles.
1. Nicastrin is required for Presenilin-mediated transmembrane cleavage in Drosophila
2. Activity-dependent isolation of the presenilin- -secretase complex reveals nicastrin and a substrate
3. aph-1 and pen-2 Are Required for Notch Pathway Signaling, γ-Secretase Cleavage of βAPP, and Presenilin Protein Accumulation
4. aph-2 encodes a novel extracellular protein required for GLP-1-mediated signaling
5. APH-1 is a multipass membrane protein essential for the Notch signaling pathway in Caenorhabditis elegans embryos
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3. γ-Secretase-Dependent Proteolysis of Transmembrane Domain of Amyloid Precursor Protein: Successive Tri- and Tetrapeptide Release in Amyloidβ-Protein Production;International Journal of Alzheimer's Disease;2012
4. PI3K inhibition causes the accumulation of ubiquitinated presenilin 1 without affecting the proteasome activity;Biochemical and Biophysical Research Communications;2010-01
5. Glu332 in the Nicastrin Ectodomain Is Essential for γ-Secretase Complex Maturation but Not for Its Activity;Journal of Biological Chemistry;2008-07
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