Intrauterine exposure to diethylhexyl phthalate disrupts gap junctions in the fetal rat testis

Author:

Di Lorenzo Mariana,Winge Sofia Boeg,Svingen Terje,De Falco Maria,Boberg Julie

Funder

Danish Environmental Protection Agency

University of Naples

Publisher

Elsevier BV

Subject

Health, Toxicology and Mutagenesis,Toxicology,Applied Microbiology and Biotechnology

Reference53 articles.

1. Use of the Adverse Outcome Pathway (AOP) framework to evaluate species concordance and human relevance of dibutyl phthalate (DBP)-induced male reproductive toxicity;Arzuaga;Reprod. Toxicol.,2019

2. Pathogenesis of male reproductive tract lesions from gestation through adulthood following in utero exposure to di(nbutyl)phthalate;Barlow;Toxicol. Pathol.,2003

3. Connexin43 gene expression and regulation in the rodent seminiferous epithelium;Batías;J. Histochem. Cytochem.,2000

4. Simvastatin and dipentyl phthalate lower ex vivo testicular testosterone production and exhibit additive effects on testicular testosterone and gene expression via distint mechanistic pathways in the fetal rat;Beverly;Toxicol. Sci.,2014

5. Early testicular effects in rats perinatally exposed to DEHP in combination with DEHA-apoptosis assessment and immunohistochemical studies;Borch;Reprod. Toxicol.,2005

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