Compensatory mutations in the matrix protein of viral hemorrhagic septicemia virus (VHSV) genotype IVa in response to artificial mutation of two amino acids (D62A E181A)-Reference-Cited by-同舟云学术

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Compensatory mutations in the matrix protein of viral hemorrhagic septicemia virus (VHSV) genotype IVa in response to artificial mutation of two amino acids (D62A E181A)

Published:2023-03 Issue: Volume:326 Page:199067
ISSN:0168-1702
Container-title:Virus Research
language:en
Short-container-title:Virus Research

Author:

Kim So Yeon,Kwak Jun Soung,Jung Wonyeong,Kim Min Sun,Kim Ki Hong^ORCID

Funder

Ministry of Science, ICT and Future Planning

National Research Foundation of Korea

Korea Ministry of Science and ICT

Publisher

Elsevier BV

Subject

Cancer Research,Infectious Diseases,Virology

Reference41 articles.

1. A single amino acid change in the non-structural NV protein impacts the virulence phenotype of Viral hemorrhagic septicemia virus in trout;Baillon;J. Gen. Virol.,2017

2. The glycoprotein of viral hemorrhagic septicemia virus (VHSV): antigenicity and role in virulence;Bearzotti;Veterinary Res.,1995

3. Identification of a pH-sensitive switch in VSV-G and a crystal structure of the G pre-fusion state highlight the VSV-G structural transition pathway;Beilstein;Cell Rep.,2020

4. Late budding domains and host proteins in enveloped virus release;Bieniasz;Virol,2006

5. The L-domains in M and G proteins of infectious hematopoietic necrosis virus (IHNV) affect viral budding and pathogenicity;Chen;Fish Shellfish Immunol.,2019

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