Systems genetics of influenza A virus-infected mice identifies TRIM21 as a critical regulator of pulmonary innate immune response-Reference-Cited by-同舟云学术

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Systems genetics of influenza A virus-infected mice identifies TRIM21 as a critical regulator of pulmonary innate immune response

Published:2024-04 Issue: Volume:342 Page:199335
ISSN:0168-1702
Container-title:Virus Research
language:en
Short-container-title:Virus Research

Author:

Li Zhuoyuan Alex,Bajpai Akhilesh Kumar,Wang Ruixue,Liu Yaxin,Webby Richard J.,Wilk Esther,Gu Weikuan,Schughart Klaus,Li Kui,Lu Lu

Funder

NIAID

National Health and Medical Research Council

College of Dentistry, Health Science Center, University of Tennessee

Helmholtz Association

Health Science Center, University of Tennessee

Publisher

Elsevier BV

Reference65 articles.

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2. Circadian rhythms in innate immunity and stress responses;Baxter;Immunology,2020

3. Targeting the proviral host kinase, FAK, limits influenza a virus pathogenesis and NFkB-regulated pro-inflammatory responses;Bergmann;Virology,2019

4. Host genetic variation affects resistance to infection with a highly pathogenic H5N1 influenza A virus in mice;Boon;J. Virol.,2009

5. Direct, interferon-independent activation of the CXCL10 promoter by NF-kappaB and interferon regulatory factor 3 during hepatitis C virus infection;Brownell;J. Virol.,2014

Cited by 2 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Exploring TRIM proteins’ role in antiviral defense against influenza A virus and respiratory coronaviruses;Frontiers in Cellular and Infection Microbiology;2024-07-15

2. Key roles for phosphorylation and the Coiled-coil domain in TRIM56-mediated positive regulation of TLR3-TRIF–dependent innate immunity;Journal of Biological Chemistry;2024-05

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