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Galectin-3 is involved in HIV-1 expression through NF-κB activation and associated with Tat in latently infected cells

  • Published:2019-01 Issue: Volume:260 Page:86-93
  • ISSN:0168-1702
  • Container-title:Virus Research
  • language:en
  • Short-container-title:Virus Research

Author:

Okamoto Mika,Hidaka Akemi,Toyama Masaaki,Baba Masanori

Funder

Scientific Research of Japan Society for the Promotion of Science

Publisher

Elsevier BV

Subject

Cancer Research,Infectious Diseases,Virology

Reference61 articles.

1. Administration of vorinostat disrupts HIV-1 latency in patients on antiretroviral therapy;Archin;Nature,2012

2. IκB: a specific inhibitor of the NF-κB transcription factor;Baeuerle;Science,1988

3. Permeability of the blood-brain barrier to HIV-1 Tat;Banks;Exp. Neurol.,2005

4. Galectins. Structure and function of a large family of animal lectins;Barondes;J. Biol. Chem.,1994

5. An essential role for NF-κB in preventing TNF-α-induced cell death;Beg;Science,1996

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