Zika NS2B is a crucial factor recruiting NS3 to the ER and activating its protease activity-Reference-Cited by-同舟云学术

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Zika NS2B is a crucial factor recruiting NS3 to the ER and activating its protease activity

Published:2020-01 Issue: Volume:275 Page:197793
ISSN:0168-1702
Container-title:Virus Research
language:en
Short-container-title:Virus Research

Author:

Xing Huaipeng,Xu Shan,Jia Fangfei,Yang Yang,Xu Caimin,Qin Chengfeng,Shi Lei

Funder

National Natural Science Foundation of China

CAMS Innovation Fund for Medical Sciences

Chinese Academy of Medical Sciences

Publisher

Elsevier BV

Subject

Cancer Research,Infectious Diseases,Virology

Reference47 articles.

1. Shaping the flavivirus replication complex: it is curvaceous!;Aktepe;Cell. Microbiol.,2018

2. Inherited microcephaly exposes Zika culprit;Ash;Science,2017

3. Biochemistry and molecular biology of flaviviruses;Barrows;Chem. Rev.,2018

4. Arsenite-induced stress granule formation is inhibited by elevated levels of reduced glutathione in West Nile virus-infected cells;Basu;PLoS Pathog.,2017

5. An update on Zika virus infection;Baud;Lancet,2017

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1. Dengue virus non-structural protein 3 inhibits mitochondrial respiration by impairing complex I function;mSphere;2024-07-30

2. The NS2B-PP1α-eIF2α axis: Inhibiting stress granule formation and Boosting Zika virus replication;PLOS Pathogens;2024-06-27

3. Japanese encephalitis virus inhibits superinfection of Zika virus in cells by the NS2B protein;Journal of Virology;2024-03-19

4. Investigating the folding dynamics of NS2B protein of Zika virus;Virology;2023-07

5. Zika virus: Critical crosstalk between pathogenesis, cytopathic effects, and macroautophagy;Journal of Cellular Biochemistry;2023-06-19

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