Long-term anti-CD4 treatment of MRL/Ipr mice ameliorates immunopathology and lymphoproliferation but fails to suppress rheumatoid factor production

Author:

O'Sullivan Frank X.,Ray Carla J.,Takeda Yoshihiko,Sharp Gordon C.,Walker Sara E.

Publisher

Elsevier BV

Subject

Immunology,Pathology and Forensic Medicine,Immunology and Allergy

Reference36 articles.

1. Autoimmunity and lymphoproliferation: Induction by mutant gene Ipr, and acceleration by a male-associated factor in strain BXSB mice;Murphy,1978

2. Spontaneous immune lupus-like syndromes: Clinical and immunopathological manifestations in several strains;Andrews;J. Exp. Med.,1978

3. A spontaneous rheumatoid arthritis-like disease in MRL/l mice;Hang;J. Exp. Med.,1982

4. Etiopathogenesis of the rheumatoid arthritis-like disease in MRL/l mice. I. The histomorphologic basis of joint destruction;O'Sullivan;Arthritis Rheum.,1985

5. Abnormalities induced by the mutant gene Ipr: Expansion of a unique lymphocyte subset;Morse;J. Immunol.,1982

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