Absence of glutamic acid decarboxylase antibodies in childhood epilepsies
Author:
Publisher
Elsevier BV
Subject
Neurology (clinical),Developmental Neuroscience,Neurology,Pediatrics, Perinatology and Child Health
Reference19 articles.
1. Two human glutamate decarboxylases, 65-kDa GAD and 67-kDa GAD, are each encoded by a single gene;Bu;Proc Natl Acad Sci U S A,1992
2. Two forms of the gamma-aminobutyric acid synthetic enzyme glutamate decarboxylase have distinct intraneuronal distributions and cofactor interactions;Kaufman;J Neurochem,1991
3. Inhibition of gamma-aminobutyric acid synthesis by glutamic acid decarboxylase autoantibodies in stiff-man syndrome;Dinkel;Ann Neurol,1998
4. Antibodies to a Mr-64,000 islet cell protein in Swedish children with newly diagnosed type 1 (insulin-dependent) diabetes;Christie;Diabetologia,1988
5. 64,000 Mr autoantibodies as predictors of insulin-dependent diabetes;Atkinson;Lancet,1990
Cited by 3 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Evaluation of Glutamic Acid Decarboxylase Antibody Levels in Patients with Juvenile Myoclonic Epilepsy and Mesial Temporal Lobe Epilepsy with Hippocampal Sclerosis;Noro Psikiyatri Arsivi;2016-08-23
2. No association of anti-GM1 and anti-GAD antibodies with juvenile myoclonic epilepsy: A pilot study;Seizure;2005-07
3. Antiphospholipid and glutamic acid decarboxylase antibodies in patients with focal epilepsy;Neurology;2004-02-09
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