Iron chelation mitigates mitochondrial dysfunction and oxidative stress by enhancing nrf2-mediated antioxidant responses in the renal cortex of a murine model of type 2 diabetes-Reference-Cited by-同舟云学术

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Iron chelation mitigates mitochondrial dysfunction and oxidative stress by enhancing nrf2-mediated antioxidant responses in the renal cortex of a murine model of type 2 diabetes

Published:2024-09 Issue: Volume:78 Page:101937
ISSN:1567-7249
Container-title:Mitochondrion
language:en
Short-container-title:Mitochondrion

Author:

Peña-Montes Donovan J.,Huerta-Cervantes Maribel,Riveros-Rosas Héctor,Manzo-Avalos Salvador,Aguilera-Méndez Asdrubal,Huerta Miguel,Trujillo Xóchitl,Cortés-Rojo Christian,Montoya-Pérez Rocío,Salgado-Garciglia Rafael,Saavedra-Molina Alfredo

Funder

Universidad Michoacana de San Nicolás de Hidalgo

Publisher

Elsevier BV

Reference75 articles.

1. Overview of the burden of chronic kidney disease in Mexico: secondary data analysis based on the Global Burden of Disease Study 2017;Agudelo-Botero;BMJ Open,2020

2. Role of intramitochondrial nitric oxide in rat heart and kidney during hypertension;Aguilera-Aguirre;Mitochondrion,2002

3. Diabetic kidney disease: Challenges, progress, and possibilities;Alicic;Clinical Journal of the American Society of Nephrology: CJASN,2017

4. Loss of iron triggers PINK1/Parkin-independent mitophagy;Allen;EMBO Rep.,2013

5. High glucose induces phosphorylation and oxidation of mitochondrial proteins in renal tubular cells: A proteomics approach;Aluksanasuwan;Sci. Rep.,2020

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