Endoplasmic reticulum stress induced apoptosis and caspase activation is mediated through mitochondria during megakaryocyte differentiation-Reference-Cited by-同舟云学术

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Endoplasmic reticulum stress induced apoptosis and caspase activation is mediated through mitochondria during megakaryocyte differentiation

Published:2020-01 Issue: Volume:50 Page:115-120
ISSN:1567-7249
Container-title:Mitochondrion
language:en
Short-container-title:Mitochondrion

Author:

Kovuru Narasaiah,Raghuwanshi Sanjeev,Sharma Durga Shankar,Dahariya Swati,Pallepati Adithya,Gutti Ravi Kumar

Funder

Council of Scientific and Industrial Research

Department of Biotechnology

Department of Science and Technology

Indian Council of Medical Research

University Grants Commission

Government of India

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Molecular Medicine

Reference39 articles.

1. Differential regulation of the apoptotic machinery during megakaryocyte differentiation and platelet production by inhibitor of apoptosis protein Livin;Abd-Elrahman;Cell Death Dis.,2013

2. Endoplasmic reticulum and the unfolded protein response: dynamics and metabolic integration;Bravo;Int. Rev. Cell Mol. Biol.,2013

3. Role of NADPH oxidase-mediated reactive oxygen species in podocyte injury;Chen;Biomed. Res. Int.,2013

4. Compartmentalized megakaryocyte death generates functional platelets committed to caspase-independent death;Clarke;J. Cell Biol.,2003

5. Platelet formation is the consequence of caspase activation within megakaryocytes;De Botton;Blood,2002

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