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Melatonin modulates permeability transition pore and 5-hydroxydecanoate induced KATP channel inhibition in isolated brain mitochondria

  • Published:2016-11 Issue: Volume:31 Page:1-8
  • ISSN:1567-7249
  • Container-title:Mitochondrion
  • language:en
  • Short-container-title:Mitochondrion

Author:

Waseem Mohammad,Tabassum Heena,Parvez Suhel

Funder

University Grants Commission

Department of Biotechnology, Government of India

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Molecular Medicine

Reference40 articles.

1. The effect of permeability transition pore opening on reactive oxygen species production in rat brain mitochondria;Akopova;Ukr. Biokhim. Zh.,2011

2. Direct inhibition of the mitochondrial permeability transition pore: a possible mechanism responsible for anti-apoptotic effects of melatonin;Andrabi;FASEB J.,2004

3. Melatonin and ischemic stroke: mechanistic roles and action;Andrabi;Adv. Pharmacol. Sci.,2015

4. Intracellular ROS protection efficiency and free radical-scavenging activity of curcumin;Barzegar;PLoS One,2011

5. The anti-inflammatory drug, nimesulide (4-nitro-2-phenoxymethane-sulfoanilide), uncouples mitochondria and induces mitochondrial permeability transition in human hepatoma cells: protection by albumin;Berson;J. Pharmacol. Exp. Ther.,2006

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