Non-cytotoxic copper overload boosts mitochondrial energy metabolism to modulate cell proliferation and differentiation in the human erythroleukemic cell line K562-Reference-Cited by-同舟云学术

Non-cytotoxic copper overload boosts mitochondrial energy metabolism to modulate cell proliferation and differentiation in the human erythroleukemic cell line K562

Published:2016-07 Issue: Volume:29 Page:18-30
ISSN:1567-7249
Container-title:Mitochondrion
language:en
Short-container-title:Mitochondrion

Author:

Ruiz Lina M.,Jensen Erik L.,Rossel Yancing,Puas German I.,Gonzalez-Ibanez Alvaro M.,Bustos Rodrigo I.,Ferrick David A.,Elorza Alvaro A.^ORCID

Funder

Fondecyt

Cochilco–Fondecyt

UNAB

Millennium Institute of Immunology and Immunotherapy

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Molecular Medicine

Reference86 articles.

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3. Stimulation of phosphoinositide 3-kinase/Akt signaling by copper and zinc ions: mechanisms and consequences;Barthel;Arch. Biochem. Biophys.,2007

4. Chapter 12 monitoring autophagic degradation of p62/SQSTM1;Bjørkøy,2009

5. A mitochondria-K+ channel axis is suppressed in cancer and its normalization promotes apoptosis and inhibits cancer growth;Bonnet;Cancer Cell,2007

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