Chronic treatment with dapagliflozin protects against mitochondrial dysfunction in the liver of C57BL/6NCrl mice with high-fat diet/streptozotocin-induced diabetes mellitus-Reference-Cited by-同舟云学术

Chronic treatment with dapagliflozin protects against mitochondrial dysfunction in the liver of C57BL/6NCrl mice with high-fat diet/streptozotocin-induced diabetes mellitus

Published:2021-07 Issue: Volume:59 Page:246-254
ISSN:1567-7249
Container-title:Mitochondrion
language:en
Short-container-title:Mitochondrion

Author:

Belosludtsev Konstantin N.,Starinets Vlada S.,Belosludtsev Maxim N.,Mikheeva Irina B.,Dubinin Mikhail V.^ORCID,Belosludtseva Natalia V.^ORCID

Funder

Russian Science Foundation

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Molecular Medicine

Reference37 articles.

1. Mouse Models of Type 2 Diabetes Mellitus in Drug Discovery;Baribault;Methods Mol Biol.,2016

2. Transport of Ca2+ and Ca2+-dependent permeability transition in rat liver mitochondria under the streptozotocin-induced type I diabetes;Belosludtsev;Cells,2019

3. Mitochondrial Ca2+ Transport: Mechanisms, Molecular Structures, and Role in Cells;Belosludtsev;Biochemistry (Mosc).,2019

4. Diabetes Mellitus, Mitochondrial Dysfunction and Ca2+-Dependent Permeability Transition Pore;Belosludtsev;Int. J. Mol. Sci.,2020

5. Itaconic acid impairs the mitochondrial function by the inhibition of complexes II and IV and induction of the permeability transition pore opening in rat liver mitochondria;Belosludtsev;Biochimie,2020

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