Investigation of decreased availability of nitric oxide precursor as the mechanism responsible for impaired endothelium-dependent vasodilation in hypercholesterolemic patients-Reference-Cited by-同舟云学术

Investigation of decreased availability of nitric oxide precursor as the mechanism responsible for impaired endothelium-dependent vasodilation in hypercholesterolemic patients

Published:1994-03 Issue:4 Volume:23 Page:844-850
ISSN:0735-1097
Container-title:Journal of the American College of Cardiology
language:en
Short-container-title:Journal of the American College of Cardiology

Author:

Casino Philip R.,Kilcoyne Crescence M.,Quyyumi Arshed A.,Koeg Jeffrey M.,Panza Julio A.

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine

Reference42 articles.

1. Nitric oxide release accounts for the biological activity of endothelium-derived relaxing factor;Palmer;Nature,1987

2. Vascular endothelial cells synthesize nitric oxide from L-arginine;Palmer;Nature,1988

3. Effect of hypercholesterolemia on vascular reactivity in the rabbit;Verbeuren;Circ Res,1986

4. Endothelium-dependent relaxation in experimental atherosclerosis in the rabbit;Jayakody;Circ Res,1987

5. Endothelium-dependent relaxation, cholesterol content and high energy metabolite balance in Watanabe hyperlipemic rabbit aorta;Ragazzi;Atherosclerosis,1989

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