Depletion of miR-155 hinders the myofibroblast activities and reactive oxygen species generation in oral submucous fibrosis
Author:
Publisher
Elsevier BV
Subject
General Medicine
Reference37 articles.
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1. Targeting MetaLnc9/miR-143/FSCN1 axis inhibits oxidative stress and myofibroblast transdifferentiation in oral submucous fibrosis;Journal of Dental Sciences;2024-07
2. Exosomal miR-17-5p derived from epithelial cells is involved in aberrant epithelium-fibroblast crosstalk and induces the development of oral submucosal fibrosis;International Journal of Oral Science;2024-06-20
3. Crosstalk of ubiquitin system and non-coding RNA in fibrosis;International Journal of Biological Sciences;2024
4. Targeting histone deacetylase 9 represses fibrogenic phenotypes in buccal mucosal fibroblasts with arecoline stimulation;Journal of Dental Sciences;2024-01
5. Exosomal miR-17-5p derived from epithelial cells is involved in aberrant epithelium-fibroblast cross-talk and induces the development of oral submucosal fibrosis;2023-10-27
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