Both GLP-1 and GIP are insulinotropic at basal and postprandial glucose levels and contribute nearly equally to the incretin effect of a meal in healthy subjects-Reference-Cited by-同舟云学术

Both GLP-1 and GIP are insulinotropic at basal and postprandial glucose levels and contribute nearly equally to the incretin effect of a meal in healthy subjects

Published:2003-07 Issue:2-3 Volume:114 Page:115-121
ISSN:0167-0115
Container-title:Regulatory Peptides
language:en
Short-container-title:Regulatory Peptides

Author:

Vilsbøll Tina,Krarup Thure,Madsbad Sten,Holst Jens J.

Publisher

Elsevier BV

Subject

Cellular and Molecular Neuroscience,Clinical Biochemistry,Endocrinology,Physiology,Biochemistry

Reference27 articles.

1. Reduction of the incretin effect in rats by the glucagon-like peptide 1 receptor antagonist exendin (9–39) amide;Kolligs;Diabetes,1995

2. Postprandial stimulation of insulin release by glucose-dependent insulinotropic polypeptide (GIP). Effect of a specific glucose-dependent insulinotropic polypeptide receptor antagonist in the rat;Tseng;J. Clin. Invest.,1996

3. Glucagon-like peptide-1 is a physiological incretin in rat;Wang;J. Clin. Invest.,1995

4. Incretin effects of increasing glucose loads in man calculated from venous insulin and C-peptide responses;Nauck;J. Clin. Endocrinol. Metab.,1986

5. Glucagon-like peptide-1 7–36: a physiological incretin in man;Kreymann;Lancet,1987

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