The FTLD Risk Factor TMEM106B Regulates the Transport of Lysosomes at the Axon Initial Segment of Motoneurons-Reference-Cited by-同舟云学术

The FTLD Risk Factor TMEM106B Regulates the Transport of Lysosomes at the Axon Initial Segment of Motoneurons

Published:2020-03 Issue:10 Volume:30 Page:3506-3519.e6
ISSN:2211-1247
Container-title:Cell Reports
language:en
Short-container-title:Cell Reports

Author:

Lüningschrör Patrick,Werner Georg,Stroobants Stijn,Kakuta Soichiro,Dombert Benjamin,Sinske Daniela,Wanner Renate,Lüllmann-Rauch Renate,Wefers Benedikt,Wurst Wolfgang,D’Hooge Rudi,Uchiyama Yasuo,Sendtner Michael,Haass Christian,Saftig Paul,Knöll Bernd,Capell Anja,Damme Markus^ORCID

Funder

Deutsche Forschungsgemeinschaft

DFG

Munich Cluster for Systems Neurology

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference45 articles.

1. Partial Tmem106b reduction does not correct abnormalities due to progranulin haploinsufficiency;Arrant;Mol. Neurodegener.,2018

2. Mutations in progranulin cause tau-negative frontotemporal dementia linked to chromosome 17;Baker;Nature,2006

3. The frontotemporal lobar degeneration risk factor, TMEM106B, regulates lysosomal morphology and function;Brady;Hum. Mol. Genet.,2013

4. Increased expression of the frontotemporal dementia risk factor TMEM106B causes C9orf72-dependent alterations in lysosomes;Busch;Hum. Mol. Genet.,2016

5. TMEM106B, the risk gene for frontotemporal dementia, is regulated by the microRNA-132/212 cluster and affects progranulin pathways;Chen-Plotkin;J. Neurosci.,2012

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