Interplay of Staphylococcal and Host Proteases Promotes Skin Barrier Disruption in Netherton Syndrome-Reference-Cited by-同舟云学术

Interplay of Staphylococcal and Host Proteases Promotes Skin Barrier Disruption in Netherton Syndrome

Published:2020-03 Issue:9 Volume:30 Page:2923-2933.e7
ISSN:2211-1247
Container-title:Cell Reports
language:en
Short-container-title:Cell Reports

Author:

Williams Michael R.,Cau Laura,Wang Yichen,Kaul Drishti,Sanford James A.,Zaramela Livia S.,Khalil Shadi,Butcher Anna M.,Zengler Karsten,Horswill Alexander R.,Dupont Christopher L.,Hovnanian Alain,Gallo Richard L.

Funder

National Institutes of Health

Atopic Dermatitis Research Network

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference68 articles.

1. Variation in Staphylococcus aureus colonization in relation to disease severity in adults with atopic dermatitis during a five-month follow-up;Alsterholm;Acta Derm. Venereol.,2017

2. New role for LEKTI in skin barrier formation: label-free quantitative proteomic identification of caspase 14 as a novel target for the protease inhibitor LEKTI;Bennett;J. Proteome Res.,2010

3. Netherton syndrome: disease expression and spectrum of SPINK5 mutations in 21 families;Bitoun;J Invest Dermatol,2002

4. Elastase 2 is expressed in human and mouse epidermis and impairs skin barrier function in Netherton syndrome through filaggrin and lipid misprocessing;Bonnart;J. Clin. Invest.,2010

5. Kallikrein 5 induces atopic dermatitis-like lesions through PAR2-mediated thymic stromal lymphopoietin expression in Netherton syndrome;Briot;J. Exp. Med.,2009

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