LncRNA HOTAIR Enhances the Androgen-Receptor-Mediated Transcriptional Program and Drives Castration-Resistant Prostate Cancer-Reference-Cited by-同舟云学术

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LncRNA HOTAIR Enhances the Androgen-Receptor-Mediated Transcriptional Program and Drives Castration-Resistant Prostate Cancer

Published:2015-10 Issue:1 Volume:13 Page:209-221
ISSN:2211-1247
Container-title:Cell Reports
language:en
Short-container-title:Cell Reports

Author:

Zhang Ali,Zhao Jonathan C.,Kim Jung,Fong Ka-wing,Yang Yeqing Angela,Chakravarti Debabrata,Mo Yin-Yuan,Yu Jindan

Funder

NIH

U.S. Department of Defense

American Cancer Society

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference31 articles.

1. Androgen receptor gene expression in prostate cancer is directly suppressed by the androgen receptor through recruitment of lysine-specific demethylase 1;Cai;Cancer Cell,2011

2. Genistein inhibits prostate cancer cell growth by targeting miR-34a and oncogenic HOTAIR;Chiyomaru;PLoS ONE,2013

3. LincRNA-p21 activates p21 in cis to promote Polycomb target gene expression and to enforce the G1/S checkpoint;Dimitrova;Mol. Cell,2014

4. Integrative genomic analyses reveal clinically relevant long noncoding RNAs in human cancer;Du;Nat. Struct. Mol. Biol.,2013

5. The mutational landscape of lethal castration-resistant prostate cancer;Grasso;Nature,2012

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