Sustained E2F-Dependent Transcription Is a Key Mechanism to Prevent Replication-Stress-Induced DNA Damage
Author:
Publisher
Elsevier BV
Subject
General Biochemistry, Genetics and Molecular Biology
Reference27 articles.
1. Cell cycle checkpoint signaling through the ATM and ATR kinases;Abraham;Genes Dev.,2001
2. Chk1 inhibits E2F6 repressor function in response to replication stress to maintain cell-cycle transcription;Bertoli;Curr. Biol.,2013
3. Control of cell cycle transcription during G1 and S phases;Bertoli;Nat. Rev. Mol. Cell Biol.,2013
4. Maintaining genome stability at the replication fork;Branzei;Nat. Rev. Mol. Cell Biol.,2010
5. Distinct but concerted roles of ATR, DNA-PK, and Chk1 in countering replication stress during S phase;Buisson;Mol. Cell,2015
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